Alzheimer ta cuta, da cututtuka da kuma mataki

Wannan cuta daukan kan musamman muhimmancin a kasashen da yawan karuwar yawan mazan mutane. Alzheimer ta cuta, da bayyanar cututtuka da wanda aka bayyana a farkon karni na 20th, shi ne har yanzu sun fi mayar da m kuma har yanzu m. Sunansa yana binta bashin da Jamusanci likita wanda farko aka bayyana shi. A Sanadin cutar ba gaba daya da aka sani. Its ci gaban da ake dangantawa da pathological tafiyar matakai a cikin kwakwalwa nama, abu don ta atrophy da total ci gaban gigin-tsufa, ko gigin-tsufa.

Masana kimiyya sun gano cewa Alzheimer ta cuta, da bayyanar cututtuka suna lalacewa ta hanyar canje-canje da fari, a cikin cerebral bawo. Akwai ragi a cikin kwakwalwa nama na acetylcholine - matsakanci, samar da alaka tsakanin neurons. Kansu kusa neurons an samu insoluble plaques hada da mahaukaci gina jiki beta-amyloid. An tabbatar da cewa sun hankali sa da mutuwar neurons. Kwakwalwa tsokoki aka gano neurofibrillary tangles, kafa tare da sa hannu na tau gina jiki. Darajar saukar a ci gaba da cututtuka na pathological siffofin apolipoprotein E, wanda kara habaka samuwar amyloid.

A sakamakon duk wadannan matakai a cikin kwakwalwa nama rage yawan kullum aiki neurons karye dangane therebetween. Akwai Aljihuna na yaxuwa atrophy, da kuma kowane irin shafi tunanin mutum aiki na marasa lafiya an keta.

Main, da wanda hade da ci gaban da cuta - wannan shi ne da shekaru. Alzheimer ta cuta, inda bayyanar cututtuka faruwa hankali da kuma ci gaba sannu a hankali a kan fiye da shekaru 10, tare da mazan mutane. A talakawan shekaru lokuta ne 54-56 shekaru. Kamar yadda muka tsufa, da yawan progressively kara.

Sakamako a kan Alzheimer ta cutar hereditary, amma ba dukkan lokuta ake dangantawa da shi. Iyalinmu siffofin da cutar ne dogara a kan pathological gene a 21 guda biyu chromosomes, wanda za a iya gaji. Da suka ci gaba a wani ƙaramin shekaru, bayan 40-50 shekaru. An yi imani da cewa idan akwai wani mutum da Alzheimer ta cuta, hadarin da ya faru an karu da sau da yawa a tsakanin dangi.

Alzheimer ta cuta, da mataki na kwarara

Bari mu bincika abin da ya canjãwa hanyar Alzheimer ta cutar. Its bayyanar cututtuka dangantawa da rushewa daga cikin al'ada jihar na kwakwalwa nama, shafi duk iri hikimar haƙƙoƙin mallaka. Ware 4, saukarwa da cutar:

Mataki na 1. preddementsii.

Da farko, akwai iya zama qananan take hakki. Marasa lafiya tabarbarewar haddacen sabon bayani, shan wahala daga hankali. Man zama apathetic. lokacin da yake magana da wahala ance kalmomi. Gaji da damuwa da fahimta da bayanai. Irin wannan yanayin zai iya dage don har zuwa shekaru 10.

2. Early gigin-tsufa.

A hankali memory deficits muni. Yana slows saukar. Rage ƙamus. Akwai take hakki na al'ada da fasaha. A haƙuri da wuya a sa a kan, rubuta, ka yi al'ada iyali chores.

3. yãfẽwa gigin-tsufa.

A yanayin da marasa lafiya worsens progressively, to cuta na magana da aka kara wa wani take hakkin rubuce-rubuce da kuma numeracy. Marasa lafiya suna ba daidaitacce a lokacin da sarari, ba ya gane dangi, da saba kewaye. Yana iya bayyana urinary incontinence. Iya zama tsokana, rũɗi. Wani lokaci akwai wani sha'awar ga vagrancy.

4. Mai tsananin gigin-tsufa.

A wannan mataki, marasa lafiya ne duka-duka da dogara a kan wasu. Su magana an kusan gaba daya rasa, ba su tuna da sunansa, da kanta, ba zai iya yi cikin sauki ayyuka. Yawancin su kwanta a fetal matsayi. A hankali da suka ci gaba cachexia, kuma su mutu daga ciwon huhu, ko shiga sauran cututtuka.

Abin baƙin ciki, to date, Alzheimer ta cuta, ta mataki, sannu-sannu wucewa cikin juna, ya ƙare da mutuwar haƙuri.

A halin yanzu gudanar da bincike don gano tasiri jiyya ga wannan cuta a duniya. Mafi yawa daga cikinsu a gwajinsu, kuma, ba shakka, a kan lokaci, bil'adama za su lashe da wannan cuta.